{"id":33843,"date":"2023-09-09T20:08:36","date_gmt":"2023-09-09T20:08:36","guid":{"rendered":"https:\/\/danielsmarketingblc.com\/CuratedProducts\/?p=33843"},"modified":"2023-09-09T20:08:37","modified_gmt":"2023-09-09T20:08:37","slug":"study-could-pave-the-method-to-recent-treatments-for-individuals-with-compromised-immune-systems","status":"publish","type":"post","link":"https:\/\/danielsmarketingblc.com\/CuratedProducts\/2023\/09\/09\/study-could-pave-the-method-to-recent-treatments-for-individuals-with-compromised-immune-systems\/","title":{"rendered":"Study could pave the method to recent treatments for individuals with compromised immune systems"},"content":{"rendered":"<p><\/p>\n<div id=\"body-4af7bfb1-b586-4bd4-a0d1-d9362e96ced6\" itemprop=\"articleBody\">\n            <span itemprop=\"author\" itemscope=\"\" itemtype=\"http:\/\/schema.org\/Organization\"><meta itemprop=\"name\" content=\"News Medical\"><meta itemprop=\"url\" content=\"https:\/\/www.news-medical.net\/\"><\/span><\/p>\n<p>Latest research led by Queen\u2019s University Belfast has made a breakthrough in the sphere of microbiology, which could lead on to the event of recent treatments for individuals with compromised immune systems, resembling those with cystic fibrosis.<\/p>\n<figure class=\"contentImage\"><span itemprop=\"image\" itemscope=\"\" itemtype=\"https:\/\/schema.org\/ImageObject\"><meta itemprop=\"url\" content=\"https:\/\/d2jx2rerrg6sh3.cloudfront.net\/images\/news\/ImageForNews_758482_16942351056975668.jpg\"><meta itemprop=\"width\" content=\"1600\"><meta itemprop=\"height\" content=\"767\"><meta itemprop=\"caption\" content=\"Study could pave the way to new treatments for people with compromised immune systems\"><span itemprop=\"thumbnail\" itemscope=\"\" itemtype=\"https:\/\/schema.org\/ImageObject\"><meta itemprop=\"url\" content=\"https:\/\/d2jx2rerrg6sh3.cloudfront.net\/image-handler\/ts\/20230909125157\/ri\/200\/src\/images\/news\/ImageForNews_758482_16942351056975668.jpg\"><meta itemprop=\"width\" content=\"200\"><meta itemprop=\"height\" content=\"96\"><\/span><\/span><figcaption class=\"imageCaption\">\n<p style=\"text-align: right;\"><em><span style=\"color:#4e5f70;\">Image Credit: Queen\u2019s University Belfast<\/span><\/em><\/p>\n<\/figcaption><\/figure>\n<p><span data-contrast=\"none\">To conduct their study the researchers checked out the bacterium <em>Achromobacter<\/em> <span data-contrast=\"none\">which may cause chronic lung infection and tissue damage within the airways.<\/span><\/span><\/p>\n<p><span data-contrast=\"none\">The study reveals how this bacterium <span data-contrast=\"none\">overcomes the body\u2019s immune defenses to multiply and proceed to grow.<\/span><\/span><\/p>\n<p><span data-contrast=\"auto\">The findings have been published in <span data-contrast=\"auto\"><em>Cell Reports<\/em>.<\/span><\/span><\/p>\n<p>Professor Miguel A. Valvano, Chair in Microbiology and Infectious Diseases on the Wellcome-Wolfson Institute for Experimental Medicine (WWIEM) at Queen\u2019s University Belfast and lead researcher on the study, e<span data-contrast=\"auto\">xplains: \u201c<\/span><em><span data-contrast=\"auto\">Achromobacter<\/span><\/em> <span data-contrast=\"auto\">bacteria may cause chronic and potentially severe infections.<\/span><span data-contrast=\"none\"> Nevertheless, until now, how this opportunistic bacterium interacts with the human immune system has been poorly understood.<\/span>\u00a0<\/p>\n<p><span data-contrast=\"none\">&#8220;These bacteria resist the motion of multiple antibiotics; due to this fact, infection by these microorganisms could be very difficult to treat by conventional therapies, especially in people living with cystic fibrosis or other immunocompromising conditions, resembling patients on chemotherapy.\u201d\u00a0<\/span><\/p>\n<p><!-- end mobile middle mrec --><\/p>\n<p><span data-contrast=\"none\">The research was led by scientists from the Valvano Group within the WWIEM at Queen\u2019s. The research team includes Dr Keren Turton, Ms Hannah Parks and Ms Paulina Zarodkiewicz, and was conducted in collaboration with Dr Rebecca Coll and Dr Rebecca Ingram, also within the WWIEM, and Professor Clare Bryant from the University of Cambridge.<\/span><\/p>\n<p><span data-contrast=\"none\">The team discovered that after being engulfed by the body\u2019s immune cells (macrophages), these bacteria can survive inside cells using a specialized protein complex (called type III secretion system) to deploy molecules that induce the death of immune cells. Self-destruction of immune cells sounds an alarm that ends in the recruitment of other immune cells to fight off invaders.<\/span><\/p>\n<p><span data-contrast=\"none\">Nevertheless, immune cells deficient in two of the inflammation sensors, called NLRC4 and NLRP3, don&#8217;t die, suggesting that these two sensors are required for the popularity of the pathogen.<\/span><\/p>\n<p><span data-contrast=\"none\">The researchers observed that <em>Achromobacter<\/em><span data-contrast=\"none\"> infection leads to wreck in lung structure and causes severe illness if the specialized secretory pathway is functional, but not if bacteria carry mutations within the secretion system.<\/span><\/span><\/p>\n<p><span data-contrast=\"none\">This demonstrates that the macrophages\u2019 self-destruct alarm is triggered by the kind III secretory system pathway but that this inflammatory response is insufficient for the immune system to defeat the bacteria.<\/span><\/p>\n<p><span data-contrast=\"none\">The following stage of the research is to find out what other virulence proteins are within the <em>Achromobacter<\/em><span data-contrast=\"none\"> armamentarium, helping it survive and invade other cell types within the body. The kind III secretion system or other proteins may very well be useful for developing novel treatments.<\/span><\/span><\/p>\n<p><span data-contrast=\"none\">This research was funded by the US Cystic Fibrosis Foundation.<\/span><\/p>\n<div id=\"sources\" class=\"content-source\">\n<p>Source:<\/p>\n<div class=\"content-src-value\">\n<p><a href=\"https:\/\/www.qub.ac.uk\/News\/Allnews\/featured-research\/new-bacteria-discovery-better-treatments-people-cystic-fibrosis.html\" target=\"_blank\" rel=\"nofollow noopener\">Queen&#8217;s University Belfast<\/a><\/p>\n<\/div>\n<p>Journal reference:<\/p>\n<div class=\"content-src-value\">\n<p>Turton, K., <em>et al<\/em>. (2023) The Achromobacter type 3 secretion system drives pyroptosis and immunopathology via independent activation of NLRC4 and NLRP3 inflammasomes. <em>Cell Reports<\/em>. <a href=\"https:\/\/doi.org\/10.1016\/j.celrep.2023.113012\" target=\"_blank\" rel=\"nofollow noopener\">doi.org\/10.1016\/j.celrep.2023.113012<\/a>.<\/p>\n<\/div>\n<\/div><\/div>\n","protected":false},"excerpt":{"rendered":"<p>Latest research led by Queen\u2019s University Belfast has made a breakthrough in the sphere of microbiology, which could lead on to the event of recent treatments for individuals with compromised immune systems, resembling those with cystic fibrosis. Image Credit: Queen\u2019s University Belfast To conduct their study the researchers checked out the bacterium Achromobacter which may 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